COBRET Laëtitia

By Isabelle Frapart on 22 février 2019

 

mail : laetitia.cobret[remplacer_par_at]cnrs-orleans.fr

Téléphone : 02.38.25.55.75

Ingénieure dans l’équipe “NEURIT : Neurobiologie des récepteurs et innovations thérapeutiques”


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12 documents

Article dans une revue8 documents

  • Chayma El Khamlichi, Laetitia Cobret, Séverine Morisset-Lopez, Séverine Morisset-Lopez. BRET Analysis of GPCR Dimers in Neurons and Non-Neuronal Cells: Evidence for Inactive, Agonist, and Constitutive Conformations. International Journal of Molecular Sciences, 2021, 22 (19), pp.10638. ⟨10.3390/ijms221910638⟩. ⟨hal-03436322⟩
  • Anthony Guillemain, Yousra Laouarem, Laetitia Cobret, Dora Štefok, Wanyin Chen, et al.. LINGO family receptors are differentially expressed in the mouse brain and form native multimeric complexes. FASEB Journal, 2020, pp.1-13. ⟨10.1096/fj.202000826R⟩. ⟨hal-02943248⟩
  • Claire Jumeau, Fawaz Awad, Eman Assrawi, Laetitia Cobret, Philippe Duquesnoy, et al.. Expression of SAA1, SAA2 and SAA4 genes in human primary monocytes and monocyte-derived macrophages. PLoS ONE, 2019, 14 (5), pp.e0217005. ⟨10.1371/journal.pone.0217005⟩. ⟨hal-02147656⟩
  • Sylvain Normand, Nadine Waldschmitt, Andre Neerincx, Ruben Julio Martinez-Torres, Camille Chauvin, et al.. Proteasomal degradation of NOD2 by NLRP12 in monocytes promotes bacterial tolerance and colonization by enteropathogens. Nature Communications, 2018, 9, pp.5338. ⟨10.1038/s41467-018-07750-5⟩. ⟨hal-01976051⟩
  • Fawaz Awad, Eman Assrawi, Claire Jumeau, Sophie Georgin-Lavialle, Laetitia Cobret, et al.. Impact of human monocyte and macrophage polarization on NLR expression and NLRP3 inflammasome activation. PLoS ONE, 2017, 12 (4), pp.e0175336. ⟨10.1371/journal.pone.0175336⟩. ⟨hal-01509838⟩
  • Wissem Deraredj Nadim, Séverine Chaumont-Dubel, Fahima Madouri, Laetitia Cobret, Marie-Ludivine de Tauzia, et al.. Physical interaction between neurofibromin and serotonin 5-HT 6 receptor promotes receptor constitutive activity. Proceedings of the National Academy of Sciences of the United States of America, 2016, 113 (43), pp.12310 - 12315. ⟨10.1073/pnas.1600914113⟩. ⟨hal-01407899⟩
  • L Cobret, M L de Tauzia, J Ferent, E Traiffort, I Hénaoui, et al.. Targeting the cis-dimerization of LINGO-1 with low MW compounds affects its downstream signalling. British Journal of Clinical Pharmacology, 2015, 172 (3), pp.841-856. ⟨10.1111/bph.12945⟩. ⟨hal-01170735⟩
  • Denis Saulnier, Sophie de Decker, Philippe Haffner, Laetitia Cobret, Maeva Robert, et al.. A Large-Scale Epidemiological Study to Identify Bacteria Pathogenic to Pacific Oyster Crassostrea gigas and Correlation Between Virulence and Metalloprotease-like Activity. Microbial ecology, 2010, 59 (4), pp.787-798. ⟨10.1007/s00248-009-9620-y⟩. ⟨hal-02114177⟩

Communication dans un congrès1 document

  • Eman Assrawi, Camille Louvrier, Clémence Lepelletier, Sophie Georgin-Lavialle, Jean-David Bouaziz, et al.. Mutation en mosaïque de NLRP3 dans des urticaires neutrophiliques avec fièvre : une nouvelle entité.. Journées dermatologiques de Paris, Dec 2019, Paris, France. ⟨inserm-03945084⟩

Poster3 documents

  • Eman Assrawi, Fawaz Awad, Claire Jumeau, Sylvie Odent, Veronique Despert, et al.. The NLRP3 p.A441V mutation in cryopyrin-associated periodic syndrome pathogenesis: functional consequences, phenotype-genotype correlations and evidence for a founder effect. ISSAID, Mar 2019, Genes, Italy. ⟨inserm-03952891⟩
  • Claire Jumeau, Fawaz Awad, Eman Assrawi, Laetitia Cobret, Philippe Duquesnoy, et al.. Expression des gènes SAA par les monocytes et macrophages humains. Nouvelle Société Francophone de l’Athérosclérose, Jun 2019, Biarritz France, France. ⟨inserm-03923123⟩
  • Hélène Bénédetti, Fabienne Godin, Michel Doudeau, Béatrice Vallée, Marie-Ludivine De Tauzia, et al.. LINGO-1, a protein involved in various neurodevelopmental processes, interacts with neurofibromin (Nf1), the protein responsible for neurofibromatosis type I: molecular studies and functional implications. 10 th FENS (Forum of Neuroscience), Jul 2016, Copenhague, Denmark. ⟨hal-02453487⟩

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